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oklahomaisforredneckjerks@yahoo.com



Re: "Bartonella may occasionally be transmitted by ticks"

dali wrote:
> If Bb does interfere with the immune system we would be talking about
> slow, wasting away diseases, such as..

Look, for one thing the thread is about Bartonella, and the FACT that
other than a VERY dubious report, there is LITTLE OR NO evidence that
Bartonella HENSALAE is of ANY clinical significance in Lyme patients.

The FACT is that Bartonella is found in a HIGH PERCENTAGE of COMPLETELY
HEALTHY people (over 80% of cat owners for example including a high
percentage who report ZERO symptoms). The bulk of the evidence is that,
other than in immunoCOMPROMISED patients (HIV patients for the most
part) and those who have had their spleens removed, it just isn't a
significant clinical entity.

AND, Bb is NOT an immunoCOMPROMISING disease, though WITHOUT DISPUTE it
affects the immune system in a variety of ways--pertaining to the
immune response to Bb, NOT GENERALLY like AIDS, thus Lyme CLEARLY is
NOT an immunocompromising disease or it would result in Lyme patients
experiencing opportunistic diseases just like AIDS patients.

Again, NO DISPUTE, that Lyme (Bb) JUST LIKE ALL INFECTIOUS AGENTS,
affects the immune response, WHICH IS NOT THE SAME AS COMPROMISING OR
"DISABLING" THE IMMUNE RESPONSE GENERALLY.

As to Lyme causing wasting type symptoms and other chronic effects, no
doubt, it is well documented.

But what does that prove about Bb and the immune system? In fact, the
plaquing seen by Miklossey may well be caused by an OVERACTIVE immune
system response--an auto immune type response.

So, Dali, this proves nothing, salvador my witless friend.

Nice try.

> Journal of Alzheimer's Disease Vol 6 No. 6 Pages 639-649 Dec. 2004
>
> Judith Miklossy, Kamel Khalili, Lise Gern, Rebecca L. Ericson, Pushpa
> Darekar, Lorie Bolle, Jean Hurlimann, Bruce J. Paster
>
> Borrelia burgdorferi persists in the brain in chronic Lyme
> neuroborreliosis and may be associated with Alzheimer's disease
>
> Abstract: The cause, or causes, of the vast majority of Alzheimer's
> disease cases are unknown. A number of contributing factors have been
> postulated, including infection. It has long been known that the
> spirochete Treponema pallidum, which is the infective agent for
> syphilis, can in its late stages cause dementia, chronic inflammation,
> cortical atrophy and amyloid deposition. Spirochetes of unidentified
> types and strains have previously been observed in the blood, CSF and
> brain of 14 AD patients tested and absent in 13 controls. In three of
> these AD cases spirochetes were grown in a medium selective for
> Borrelia burgdorferi. In the present study, the phylogenetic analysis
> of these spirochetes was made. Positive identification of the agent as
> Borrelia burgdorferi sensu stricto was based on genetic and molecular
> analyses. Borrelia antigens and genes were co-localized with
> beta-amyloid deposits in these AD cases.
>
> http://www.canlyme.com/alzjournal6.html




Old Post 11-14-05 10:54 PM
   Edit/Delete IP: Logged
Martijn



Re: "Bartonella may occasionally be transmitted by ticks"
oklahomaisforredneckjerks@yahoo.com wrote:

> There really is no evidence that bartonella HENSELAE is a human health
> problem EXCEPT in spenectomized and immundeficient patients.
>
> The ONE article by Eskow and Mordecai was laughable AND any true Lyme
> patient advocate would realize the simple problem with it: they tested
> patients who had been "previously treated" for Lyme (treated a la
> Steere) and found bartonella HENSELAE.
>
> Now they treated those same patients with CLAFORAN (a drug that does
> WONDERS for Lyme) and not surprisingly the patients showed
> improvement--as almost all LYME patients treated with Claforan
> INCLUDING THOSE PREVIOUSLY TREATED (a la Steere), and their conclusion:
> bartonella was the problem.
>
> An absurd and poorly supported conclusion.
>
> A paper co written by Eli Mordecai, OWNER of Mdlabs who CLEARLY has an
> interest in selling the bartonella testing (and has been well known to
> minimize the seriousness of Lyme alone--he really is quite skeptical
> about the notion of "chronic Lyme" AND his "lab" SELLS some VERY
> questionable testing which has not ever been properly validated).

Thank you for these informations! (there are still people here who are
interested in Lyme-related topics)



Old Post 11-14-05 10:54 PM
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Mockingbird



Re: "Bartonella may occasionally be transmitted by ticks"

Martijn wrote:
> dali wrote: 
>
> Life is a slow, wasting away disease anyway.

was Jean Paul Sartre who said that "life is like a leak of gas from a
balloon" (or something like that, it is paraphrased before our local
janitor philosophy expert chimes in with some smart XXX correction

Speaking of leaks of gas, that's what I consider Brucie, a foul wind
blowing..




Old Post 11-14-05 10:54 PM
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Mockingbird



Re: "Bartonella may occasionally be transmitted by ticks"

dali wrote:
> oklahomaisforredneckjerks@yahoo.com wrote: 
>
>
> Dr. R. Stricker: Using the CD57 Natural Killer Cell Marker to Measure
> Treatment Progress
> http://www.ncbi.nlm.nih.gov/entrez/..ieve&db=3D=3DP=
ubMed&list_uids=3D.088407&dopt=3D=ABstract
>
> One of the difficult things about treating chronic Lyme patients is
> figuring out when to stop antibiotics, since the antibody and PCR tests
> are so unreliable. Dr. Stricker has been doing research that measures
> the CD57 Natural Killer (NK) Cell levels as a way of determining if a
> chronic lyme patient is getting better. (The CD57 Natural Killer Cell
> is an aggressive white blood cell or "lymphocyte" that seeks out
> invading germs and destroys them.) It appears that Bb and all the
> coinfections somehow disable the production of the CD57 natural killer
> cells. (The "Stricker Panel," which is ordered by many Lyme Literate
> Physicians measures this immune system marker, among others.)
> www.anapsid.org/lyme/strickerpanel.html


I will comment further in my response to this:


1: Immunol Lett 2001 Feb 1;76(1):43-8


Decreased CD57 lymphocyte subset in patients with chronic Lyme disease.



Stricker RB, Winger EE.


Department of Medicine, California Pacific Medical Center, 450 Sutter
Street,
Suite 1504, San Francisco, CA 94108, USA.


BACKGROUND:
Chronic Lyme disease (LD) is a debilitating illness caused by tickborne

infection with the spirochete Borrelia burgdorferi. Although
immunologic
abnormalities appear to play a role in this disease, specific
immunologic
markers of chronic LD have not been identified.
METHODS: We evaluated 73 patients with chronic LD for lymphocyte subset

abnormalities using flow cytometry. Of these, 53 patients had
predominant
musculoskeletal symptoms, while 20 patients had predominant neurologic
symptoms. The estimated duration of infection ranged from 3 months to
15 years,
and all patients had positive serologic tests for B. burgdorferi. Ten
patients
with acute LD (infection less than 1 month) and 22 patients with
acquired
immunodeficiency syndrome (AIDS) served as disease controls.
RESULTS:  All 31 chronic LD patients who were tested prior to
antibiotic
treatment had significantly decreased CD57 lymphocyte counts (mean,
30+/-16
cells per microl; normal, 60-360 cells per microl, P<0.001). Nineteen
of 37
patients (51%) who were tested after initiating antibiotic therapy had
decreased CD57 levels (mean, 66+/-39 cells per microl), and all five
patients
tested after completing antibiotic treatment had normal CD57 counts
(mean,
173+/-98 cells per microl). In contrast, all 10 patients with acute LD
and 82%
of AIDS patients had normal CD57 levels, and the difference between
these
groups and the pre-treatment patients with chronic LD was significant
(P<0.001). Patients with chronic LD and predominant neurologic symptoms
had
significantly lower mean CD57 levels than patients with predominant
musculoskeletal symptoms (30+/-21 vs. 58+/-37 cells per microl,
P=3D0.002). CD57
levels increased in chronic LD patients whose symptoms improved, while
patients
with refractory disease had persistently low CD57 counts. CONCLUSIONS:
A
decrease in the CD57 lymphocyte subset may be an important marker of
chronic
LD. Changes in the CD57 subset may be useful to monitor the response to
therapy
in this disease.=20


PMID: 11222912 [PubMed - indexed for MEDLINE]




Old Post 11-14-05 10:54 PM
   Edit/Delete IP: Logged
Mockingbird



Re: "Bartonella may occasionally be transmitted by ticks"

Mockingbird wrote:
> dali wrote: 
=3DPubMed&list_uids=3D.088407&dopt=3D=ABstract[vbcol=seagreen] 
>
>
> I will comment further in my response to this:
>
>
> 1: Immunol Lett 2001 Feb 1;76(1):43-8
>
>
> Decreased CD57 lymphocyte subset in patients with chronic Lyme disease.
>
>
>
> Stricker RB, Winger EE.
>
>
> Department of Medicine, California Pacific Medical Center, 450 Sutter
> Street,
> Suite 1504, San Francisco, CA 94108, USA.
>
>
> BACKGROUND:
> Chronic Lyme disease (LD) is a debilitating illness caused by tickborne
> infection with the spirochete Borrelia burgdorferi. Although
> immunologic  abnormalities appear to play a role in this disease, specific
> immunologic  markers of chronic LD have not been identified.
> METHODS: We evaluated 73 patients with chronic LD for lymphocyte subset
> abnormalities using flow cytometry. Of these, 53 patients had
> predominant musculoskeletal symptoms, while 20 patients had predominant n=
eurologic
> symptoms. The estimated duration of infection ranged from 3 months to
> 15 years, and all patients had positive serologic tests for B. burgdorfer=
i=2E Ten
> patients  with acute LD (infection less than 1 month) and 22 patients with
> acquired  immunodeficiency syndrome (AIDS) served as disease controls.
>  RESULTS:  All 31 chronic LD patients who were tested prior to
> antibiotic  treatment had significantly decreased CD57 lymphocyte counts =
(mean,
> 30+/-16  cells per microl; normal, 60-360 cells per microl, P<0.001).


> Nineteen
> of 37  patients (51%) who were tested after initiating antibiotic therapy=
had
> decreased CD57 levels (mean, 66+/-39 cells per microl),

This really makes NO sense. 31 patients had low CD 57 levels BEFORE
starting abx. Then 19 had low CD57 levels AFTER starting therapy. But
then five patients had normal CD57 counts AFTER completing it?

What about the others?

Was there any patient followed from beginning to end? The way this is
presented is MEANINGLESS.

Also: were these patients tested for OTHER things that might cause low
CD57 levels?

And where was this ever confirmed?

What were the methods?

This is an utterly meaningless jumble. Embarassing.

>and all five
> patients  tested after completing antibiotic treatment had normal CD57 co=
unts
> (mean, 173+/-98 cells per microl). In contrast, all 10 patients with acut=
e LD
> and 82% of AIDS patients had normal CD57 levels, and the difference betwe=
en
> these  groups and the pre-treatment patients with chronic LD was signific=
ant
> (P<0.001). Patients with chronic LD and predominant neurologic symptoms
> had  significantly lower mean CD57 levels than patients with predominant
> musculoskeletal symptoms (30+/-21 vs. 58+/-37 cells per microl,
> P=3D0.002). CD57  levels increased in chronic LD patients whose symptoms =
improved, while
> patients  with refractory disease had persistently low CD57 counts. CONCL=
USIONS:
> A decrease in the CD57 lymphocyte subset may be an important marker of
> chronic  LD. Changes in the CD57 subset may be useful to monitor the resp=
onse to
> therapy  in this disease.
>
>
> PMID: 11222912 [PubMed - indexed for MEDLINE]

Really Dr Stricker this means nothing presented this way! Of course, as
one of the proponents of the UTTERLY DISCREDITED theory that AIDS
originated from contaminated Polio vaccine, you just ought to be
embarassed period!




Old Post 11-14-05 10:54 PM
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Paid Steere Plant



Re: "Bartonella may occasionally be transmitted by ticks"
gotta chime in like a smart XXX-

rednecks {who, let's face it, is really mr. ed} mentioned that ~ 80% of
humans demonstrated exposure to bartonella.
well, at least that percentage demonstrate exposure to human papilloma
virus..
and fauci wants a vaccine for that, because of the resulting ~3000
cases/year of cervical cancer. while it is unlikely, but not
demonstrated, that bartonella causes such severe sequelae - the point
is that an infectious agent to which most people have experienced
exposure CAN cause dangerous sequelae in a small number of susceptible
individuals. it is erroneous to assume that commonality of exposure
equates to presumed harmlessness of the agent involved. this is not
always the case. millions of young woman will soon face mandatory (i
think) vaccination for precisely this reason. and the FACT is, very
little is known about bartonella  in humans. could it be along the same
lines as borna virus or the recently discussed spiroplasm? Probably
not, but if you just  close the door without being careful..you could
impede progress.

also, rednecks ..terrible logical error there in thinking that "if
lyme were immunocompromising, we would see {an AIDS-like syndrome}..or
something of this nature.

FACT..the immune system is much more complicated than you give it
credit for. there are multiple levels at which "compromise" can occur.
AIDS is devastating, but it is a retrovirus. lyme, the last time I
checked, is a spirochete - a bacteria. there are dozens {if not more}
of different syndromes other than aids which result in
"immunocompromise". do they all present in exactly the same fashion as
AIDS? No. lyme has been postulated  to be a more chronic, less acute
infection than full blown aids for an extreme comparison {and obviously
not as fatal}. how can you be certain that there is not similarly
"immunocompromise", of a less obvious and thus less detectable and easy
to understand level? you're comparing a dramatic illness, aids, with a
totally different entity. science is not so cut and dry. it is far more
complex than you give credit. simply making ASSumptions like this is
what has crippled lyme clinical research in humans for the past 15
years.

this is a blanket generalization - of the same sort of fallacy which
klempner employed when he so stupidly compared borellia species to
treponema. it's not an intelligent thing to say. very arrogant, and
assumes much knowledge which you simply do not possess. for shits sake,
your statements..ludicrously arrogant and insulting, really..what is
the sequence homology of the borellia genus with other genuses? you
know the answer to that. how many protein structures have been
elucidated? do we know exactly what even a FRACTION of them do? etc.

you must have been involved in the OspA debacle.

the janitor




Old Post 11-15-05 05:54 AM
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Martijn



Re: "Bartonella may occasionally be transmitted by ticks"
oklahomaisforredneckjerks@yahoo.com wrote:

> The FACT is that Bartonella is found in a HIGH PERCENTAGE of COMPLETELY
> HEALTHY people (over 80% of cat owners for example including a high
> percentage who report ZERO symptoms).

I am very interested in this. Do you have references?



Old Post 11-15-05 03:54 PM
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Mockingbird



Re: "Bartonella may occasionally be transmitted by ticks"

Paid Steere Plant wrote:
> gotta chime in like a smart XXX-
>
> rednecks {who, let's face it, is really mr. ed}

LOL Well I'll tell you this much you delusional freak, you CLEARLY are
a horse's XXX but I'm NOT "mr ed" but if you prefer to think that
you're engaging in a battle of good and evil with ed mcsweegan, and
that makes you feel important like lisa and kathLOON, then think what
you will you freaking fool!

>mentioned that ~ 80% of
> humans demonstrated exposure to bartonella.
> well, at least that percentage demonstrate exposure to human papilloma
> virus..

And the two aren't comparable. Why even bother with this nonsense
comparing apples and oranges? In fact, your original screed goes on
without noting that the article which began the thread talks about
Bartonella vinsonii subsp. arupensis, rather than bartonella HENSELAE,
and the two are NOT the same just as all borellia are NOT the same YOU
MORON!

> and fauci wants a vaccine for that, because of the resulting ~3000
> cases/year of cervical cancer. while it is unlikely, but not
> demonstrated, that bartonella causes such severe sequelae - the point
> is that an infectious agent to which most people have experienced
> exposure CAN cause dangerous sequelae in a small number of susceptible
> individuals. it is erroneous to assume that commonality of exposure
> equates to presumed harmlessness of the agent involved. this is not
> always the case.

But it IS the case with bartonella henselae, NOT that it is "harmless"
but that it is considered self limited and the value of treatment in
immunocompetent individuals and others NOT susceptible to sequelae is
highly dubious!

Still, IF I were treating a patient and they tested positive, I'd
treat--but for a REASONABLE period of time, NOT like the llmds.

>millions of young woman will soon face mandatory (i
> think) vaccination for precisely this reason. and the FACT is, very
> little is known about bartonella  in humans. could it be along the same
> lines as borna virus or the recently discussed spiroplasm? Probably
> not, but if you just  close the door without being careful..you could
> impede progress.

No one's trying to impede progress you XXXXXXX!

> also, rednecks ..terrible logical error there in thinking that "if
> lyme were immunocompromising, we would see {an AIDS-like syndrome}..
or
> something of this nature.

IF it were immunoCOMPROMISING yes you would see an AIDS like syndrome
MORON!

Because if the immune system is compromised (you actually used the word
"disabled") then there'd be nothing to fight opportunistic infection!

MORON!

> FACT..the immune system is much more complicated than you give it
> credit for.

FACT: YOU are much more simple minded than anyone can believe!

Look XXXXXXX, once again, I didn't say what you're saying I did, but
YOU did say what I'm saying YOU did and what I was responding to!

I didn't say Lyme doesn't AFFECT the immune system. In fact, I said it
did, exactly I said it did! I just said it doesn't COMPROMISE OR
DISABLE the immune system AND IF IT DID then Lymies would get
opportunistic diseases WHICH IS TRUE but since it doesn't, they don't!

>there are multiple levels at which "compromise" can occur.

YOU said DISABLE and it doesn't do that! And YOU didn't specify at what
level!

A normal person would simply accept the distinction I made and clarify.
But YOU attack in response you psychotic XXXXXXX!

> AIDS is devastating, but it is a retrovirus. lyme, the last time I
> checked, is a spirochete - a bacteria. there are dozens {if not more}
> of different syndromes other than aids which result in
> "immunocompromise".

Look MORON, the important distinction is NOT whether it is caused by a
retrovirus OR a bacteria. The important distinction is that you didn't
MEAN immunoCOMPROMISE or AS YOU ACTUALLY SAID DISABLE THE IMMUNE
SYSTEM.

You MEANT and should have said that Lyme AFFECTS the immune response.

The term IMMUNOCOMPROMISE has a specific meaning in medicine. When we
talk about "immunocompromised patients" (as being susceptible to
serious sequelae of bartonella HENSALAE, we're talking about a specific
inability of the immune system to respond to ordinary infections NOT
specific immune system abnormalities caused by Lyme which are NOT
generally immunocompromising or GENERALLY immunodisabling!

Just accept the apt distinction and move on with your miserable
pathetic excuse for a life JANITOR and stop pretending to be a
microbiologist, doctor, scientist or anything but the JANITOR MENTAL
PATIENT which is all that you really have on your pathetic waste of a
life resume!

>do they all present in exactly the same fashion as
> AIDS? No. lyme has been postulated  to be a more chronic, less acute
> infection than full blown aids for an extreme comparison {and obvious
ly
> not as fatal}. how can you be certain that there is not similarly
> "immunocompromise", of a less obvious and thus less detectable and easy
> to understand level?


Because other than the one dubious stricker report about CD57
abnormalities, most Lyme patients do NOT have any quantititive
abnormalities in their immune system and MANY have been tested and
those that DO have abnormalities are NOT attributable to Lyme but to
other conditions!

>you're comparing a dramatic illness, aids, with a
> totally different entity. science is not so cut and dry.

No I'm talking about IMMUNOCOMPROMISE no matter what the cause or your
phrase DISABLE THE IMMUNE SYSTEM the cause doesn't matter.

The distinction would be that it is NOT a GENERAL disabling but perhaps
specific abnormalities result!

> it is far more  complex than you give credit.

No IT isn't.

But YOU are!


>simply making ASSumptions like this is  what has crippled lyme clinical research in
 >humans for the past 15  years.

No, but know it alls who know NOTHING like you and blather on the
internet and would rather fight about what the meaning of the word "is"
is are a BIG part of the problem!

> this is a blanket generalization - of the same sort of fallacy which
> klempner employed when he so stupidly compared borellia species to
> treponema.

Yes that was a stupid thing to say!

By the way, Phyllis, who you praised the other day, was a "patient
representative" on the NIH chronic Lyme disease study. She signed off
on the study design, praised klempner, helped recruit for the study,
tried to silence critics who spoke out at the time, and never went on
record about the problems she later claimed she was aware of!

She's a BIG HELP!

So go on and heap praise on her. She's Neville Chamberlin and you're a
MORON!

>it's not an intelligent thing to say. very arrogant, and
> assumes much knowledge which you simply do not possess. for shits sake,
> your statements..ludicrously arrogant and insulting, really..what is
> the sequence homology of the borellia genus with other genuses? you
> know the answer to that. how many protein structures have been
> elucidated? do we know exactly what even a FRACTION of them do? etc.

Irrelevant. LYME DOES NOT CAUSE IMMUNOCOMPROMISE AS THE TERM IS USED
AND UNDERSTOOD NOR DOES IT DISABLE THE IMMUNE SYSTEM PLAIN AND SYSTEM
SHUT UP ALREADY MAKE A USEFUL DISTINCTION AND MOVE ON YOU XXXXXXX!

> you must have been involved in the OspA debacle.
>
> the janitor

You mean the MENTAL PATIENT JANITOR PSYCHOTIC XXXXXXX SHITHEAD
CHICKENSHIT MORON




Old Post 11-15-05 03:54 PM
   Edit/Delete IP: Logged
Mockingbird



Re: "Bartonella may occasionally be transmitted by ticks"
Hey brucie why don't you wax on a bit more comparing bartonella or Lyme
to human pappiloma virus and continue to misuse the phrase
"immunocompromise" and tell us about all the "PROOF" that bartonella is
such a big deal clinically???

MORON


Mockingbird wrote:
> Paid Steere Plant wrote: 
>
> LOL Well I'll tell you this much you delusional freak, you CLEARLY are
> a horse's XXX but I'm NOT "mr ed" but if you prefer to think that
> you're engaging in a battle of good and evil with ed mcsweegan, and
> that makes you feel important like lisa and kathLOON, then think what
> you will you freaking fool!
> 
>
> And the two aren't comparable. Why even bother with this nonsense
> comparing apples and oranges? In fact, your original screed goes on
> without noting that the article which began the thread talks about
> Bartonella vinsonii subsp. arupensis, rather than bartonella HENSELAE,
> and the two are NOT the same just as all borellia are NOT the same YOU
> MORON!
> 
>
> But it IS the case with bartonella henselae, NOT that it is "harmless"
> but that it is considered self limited and the value of treatment in
> immunocompetent individuals and others NOT susceptible to sequelae is
> highly dubious!
>
> Still, IF I were treating a patient and they tested positive, I'd
> treat--but for a REASONABLE period of time, NOT like the llmds.
> 
>
> No one's trying to impede progress you XXXXXXX!
> 
>
> IF it were immunoCOMPROMISING yes you would see an AIDS like syndrome
> MORON!
>
> Because if the immune system is compromised (you actually used the word
> "disabled") then there'd be nothing to fight opportunistic infection!
>
> MORON!
> 
>
> FACT: YOU are much more simple minded than anyone can believe!
>
> Look XXXXXXX, once again, I didn't say what you're saying I did, but
> YOU did say what I'm saying YOU did and what I was responding to!
>
> I didn't say Lyme doesn't AFFECT the immune system. In fact, I said it
> did, exactly I said it did! I just said it doesn't COMPROMISE OR
> DISABLE the immune system AND IF IT DID then Lymies would get
> opportunistic diseases WHICH IS TRUE but since it doesn't, they don't!
> 
>
> YOU said DISABLE and it doesn't do that! And YOU didn't specify at what
> level!
>
> A normal person would simply accept the distinction I made and clarify.
> But YOU attack in response you psychotic XXXXXXX!
> 
>
> Look MORON, the important distinction is NOT whether it is caused by a
> retrovirus OR a bacteria. The important distinction is that you didn't
> MEAN immunoCOMPROMISE or AS YOU ACTUALLY SAID DISABLE THE IMMUNE
> SYSTEM.
>
> You MEANT and should have said that Lyme AFFECTS the immune response.
>
> The term IMMUNOCOMPROMISE has a specific meaning in medicine. When we
> talk about "immunocompromised patients" (as being susceptible to
> serious sequelae of bartonella HENSALAE, we're talking about a specific
> inability of the immune system to respond to ordinary infections NOT
> specific immune system abnormalities caused by Lyme which are NOT
> generally immunocompromising or GENERALLY immunodisabling!
>
> Just accept the apt distinction and move on with your miserable
> pathetic excuse for a life JANITOR and stop pretending to be a
> microbiologist, doctor, scientist or anything but the JANITOR MENTAL
> PATIENT which is all that you really have on your pathetic waste of a
> life resume!
> 
>
>
> Because other than the one dubious stricker report about CD57
> abnormalities, most Lyme patients do NOT have any quantititive
> abnormalities in their immune system and MANY have been tested and
> those that DO have abnormalities are NOT attributable to Lyme but to
> other conditions!
> 
>
> No I'm talking about IMMUNOCOMPROMISE no matter what the cause or your
> phrase DISABLE THE IMMUNE SYSTEM the cause doesn't matter.
>
> The distinction would be that it is NOT a GENERAL disabling but perhaps
> specific abnormalities result!
> 
>
> No IT isn't.
>
> But YOU are!
>
> 
>
> No, but know it alls who know NOTHING like you and blather on the
> internet and would rather fight about what the meaning of the word "is"
> is are a BIG part of the problem!
> 
>
> Yes that was a stupid thing to say!
>
> By the way, Phyllis, who you praised the other day, was a "patient
> representative" on the NIH chronic Lyme disease study. She signed off
> on the study design, praised klempner, helped recruit for the study,
> tried to silence critics who spoke out at the time, and never went on
> record about the problems she later claimed she was aware of!
>
> She's a BIG HELP!
>
> So go on and heap praise on her. She's Neville Chamberlin and you're a
> MORON!
> 
>
> Irrelevant. LYME DOES NOT CAUSE IMMUNOCOMPROMISE AS THE TERM IS USED
> AND UNDERSTOOD NOR DOES IT DISABLE THE IMMUNE SYSTEM PLAIN AND SYSTEM
> SHUT UP ALREADY MAKE A USEFUL DISTINCTION AND MOVE ON YOU XXXXXXX!
> 
>
> You mean the MENTAL PATIENT JANITOR PSYCHOTIC XXXXXXX SHITHEAD
> CHICKENSHIT MORON




Old Post 11-24-05 03:54 PM
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