| ironjustice@aol.com 2006-10-03, 2:30 am |
| Dr. Jerome Sullivan states ferritin is a storage site for iron not
being utilized.
Iron not being utilized is therefore .. logically .. unneeded.
"Incorrect views on iron are institutionalized not only in the
medical profession, but also in industry and in government."
---Jerome Sullivan, M.D., Ph.D.
It seems in some cases ferritin promotes disease.
As in the case of arsenic .. it seems .. and also .. electromagnetic
hypersensitivity.
Arch Biochem Biophys. 2000 Oct 15;382(2):195-202. Links
Arsenic species that cause release of iron from ferritin and generation
of activated oxygen.Ahmad S, Kitchin KT, Cullen WR.
Environmental Carcinogenesis Division, US Environmental Protection
Agency, Research Triangle Park, North Carolina 27711, USA.
The in vitro effects of four different species of arsenic (arsenate,
arsenite, monomethylarsonic acid, and dimethylarsinic acid) in
mobilizing iron from horse spleen ferritin under aerobic and anaerobic
conditions were investigated. Dimethylarsinic acid (DMA(V)) and
dimethylarsinous acid (DMA(III)) significantly released iron from horse
spleen ferritin either with or without the presence of ascorbic acid, a
strong synergistic agent. Ascorbic acid-mediated iron release was
time-dependent as well as both DMA(III) and ferritin
concentration-dependent. Iron release from ferritin by DMA(III)) alone
or with ascorbic acid was not significantly inhibited by superoxide
dismutase (150 or 300 units/ml). However, the iron release was greater
under anaerobic conditions (nitrogen gas), which indicates direct
chemical reduction of iron from ferritin by DMA(III), with or without
ascorbic acid. Both DMA(V) and DMA(III)) released iron from both horse
spleen and human liver ferritin. Further, the release of ferritin iron
by DMA(III)) with ascorbic acid catalyzed bleomycin-dependent
degradation of calf thymus DNA. These results indicate that exogenous
methylated arsenic species and endogenous ascorbic acid can cause (a)
the release of iron from ferritin, (b) the iron-dependent formation of
reactive oxygen species, and (c) DNA damage. This reactive oxygen
species pathway could be a mechanism of action of arsenic
carcinogenesis in man.
PMID: 11068869 [PubMed - indexed for MEDLINE]
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EMF-cancer link: the ferritin hypothesis.
by Shao T
published in: Med Hypotheses 1993; 41 (1): 28 - 30 ( PubMed Entry )
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Who loves ya.
Tom
Jesus Was A Vegetarian!
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Man Is A Herbivore!
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DEAD PEOPLE WALKING
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