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Home > Archive > Medicine laboratory > March 2006 > Where credit is .. DUE .. / I am right again / will harris eat crow?
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Where credit is .. DUE .. / I am right again / will harris eat crow?
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| ironjustice@aol.com 2006-02-25, 8:48 pm |
| http://tinyurl.com/ckq3e
Ten years almost to the day ..
Iron supplements 'increase malaria threat' - SciDev.NetChildren at risk
of malaria are more likely to die of the disease if they are given
dietary supplements of iron and folic acid, according to a study in the
....
www.scidev.net/content/news/eng/
iron-supplements-increase-malaria-threat.cfm - 19k - Jan 19, 2006 -
And below .. the 'medical' .. take .. on it ..
Heh .. heh ..
No wonder everyone is dying at such a high rate ..
http://tinyurl.com/9z42a
<<snip>>
COMMENT:
Oh, I see. Your assertion is based on completely data-free
blue sky hypothesizing from some guy who did one study in
MICE.
Whereas the contrary evidence that iron supplementation is
good for African children in malaria epidemic areas comes
from scientists who actually fed iron to such children, and
*directly* observed the increase in health (motor and mental
development).
As I said, you're a complete moron
<<snip>>
And I would say .. the above speaker has been PROVEN to be a .. moron
...
Logic ..
Yep ..
Heh .. heh ..
Who loves ya.
Tom
Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com
Man Is A Herbivore!
http://pages.ivillage.com/ironjustice/manisaherbivore
DEAD PEOPLE WALKING
http://pages.ivillage.com/ironjustice/deadpeoplewalking
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| Manky Badger 2006-02-25, 8:48 pm |
|
<ironjustice@aol.com> wrote in message
news:1137862999.493106.254370@g43g2000cwa.googlegroups.com...
> http://tinyurl.com/ckq3e
>
> Ten years almost to the day ..
OK - this has been going on for ages.
I have to ask.
Tom - why iron ?
There's endless other medical/dietry bandwagons you could have jumped on.
Why specifically iron ?
What's the special significance - ten years - this has to be personal ?
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| mournenwould@aol.com 2006-02-25, 8:48 pm |
| Iron is constipating.
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| johngohde@naturalhealthperspective.com 2006-02-25, 8:48 pm |
|
Manky Badger wrote:
> <ironjustice@aol.com> wrote in message
> news:1137862999.493106.254370@g43g2000cwa.googlegroups.com...
>
> OK - this has been going on for ages.
> I have to ask.
>
> Tom - why iron ?
Mental illness is real.
And, Tom's signatures have been documenting that he is totally
confused. 
| |
| ironjustice@aol.com 2006-02-25, 8:48 pm |
| Once-dreaded leprosy 'replaced' by tuberculosis, say researchers
01 Aug 2005
What caused leprosy - a widely dreaded disease in medieval Europe - to
fade from the scene? By the 16th century, the scourge had practically
disappeared there.
The reason seems to be, say researchers at the Hebrew university of
Jerusalem and in London, that tuberculosis, a far more deadly disease,
overtook leprosy, killing millions throughout Europe.
Their conclusion is based upon the examination of DNA from human
remains from the ancient and medieval periods in Israel and Europe. In
these examinations, the scientists found traces of both leprosy and
tuberculosis bacteria in 42 percent of the cases.
The findings on the relationship between leprosy and tuberculosis were
reported in a recent edition of the British Royal Society Proceedings B
by Dr. Mark Spigelman, a visiting professor at the Hebrew University
Faculty of Medicine and of the university college London; Prof. Charles
Greenblatt of the Sanford F. Kuvin Center for the Study of Infectious
and Tropical Diseases at the Hebrew University's Faculty of Medicine;
and Dr. Helen Donoghue of university college London.
The earliest case of co-infection of both leprosy and tuberculosis was
found by the researchers in the DNA from a body discovered in a 1st
century CE burial cave in Jerusalem. This prompted the investigators to
re-examine DNA samples from other ancient sites that they and their
colleagues had worked on previously. In doing so, they found leprosy
and tuberculosis bacteria in remains from a 4th century CE Egyptian
shrine that was known to have been visited by lepers, from a 10th
century burial ground in Hungary, and from a Viking-age cemetery in
northern Sweden.
The conclusion drawn by the researchers from these multiple signs of
co-infection of leprosy and TB bacteria is that those with leprosy,
which was seldom fatal, were weakened to the extent that they became
highly vulnerable to the "big killer," tuberculosis. This was
exacerbated by the lives of deprivation that the lepers were forced to
live as social outcasts.
Ultimately, the scientists theorize, so many of the lepers died of
tuberculosis until there were too few of them to further spread
leprosy. TB, meanwhile, was increasingly on the rise as people in the
Middle Ages migrated to urban centers, where crowding and poor sanitary
conditions provided a fertile breeding ground for the spread of the
killer disease.
Today, tuberculosis, though curable, is still a major, long-term,
epidemic disease, with millions of new cases reported around the world
each year.
Contact: Jerry Barach
jer...@savion.huji.ac.il
972-2-588-2904
The Hebrew university of Jerusalem
http://www.huji.ac.il
---------------------------------------------------------------------------=
=AD-----
<<snip>>
The management of dietary iron can therefore be influential in aiding
the outcome of this disease.
<<snip>>
Tuberculosis (Edinb). 2004;84(1-2):110-30. Related Articles, Links
Iron, mycobacteria and tuberculosis.
Ratledge C.
Department of Biological Sciences, university of Hull, Hull HU6 7RX,
UK. c.ratle...@biosci.hull.ac.uk
The role of iron in the growth and metabolism of M. tuberculosis and
other mycobacteria is discussed in relation to the acquisiton of iron
from host sources, such as transferrin, lactoferrin and ferritin, and
its subsequent assimilation and utilization by the bacteria. Key
components involved in the acquisition of iron (as ferric ion) and its
initial transport into the mycobacterial cell are extracellular iron
binding agents (siderophores) which, in pathogenic mycobacteria, are
the carboxymycobactins and, in saprophytic mycobacteria, are the
exochelins. In both cases, iron may be transferred to an
intra-envelope, short-term storage molecule, mycobactin. For transport
across the cell membrane, a reductase is used which converts
FeIII-mycobactin to the FeII form. The ferrous ion, possibly complexed
with salicylic acid, is then shuttled across the membrane either for
direct incorporation into various porphyrins and apoproteins or, for
storage of iron within the bacterial cytoplasm, bacterioferritin. The
overall process of iron acquisition and its utilization is under very
genetic tight control. The importance of iron in the virulence of
mycobacteria is discussed in relationship to the development of
tuberculosis. The management of dietary iron can therefore be
influential in aiding the outcome of this disease. The role of the old
anti-TB compound, p-aminosalicylate (PAS), is discussed in its action
as an inhibitor of iron assimilation, together with the prospects of
being able to synthesize further selective inhibitors of iron
metabolism that may be useful as future chemotherapeutic agents.
Publication Types:
Review
Review, Tutorial
PMID: 14670352 [PubMed - indexed for MEDLINE]
------------------------------=AD------------------------------=AD---------=
----=AD-------
Int J Tuberc Lung Dis. 2005 Jan;9(1):2-9. Related Articles, Links
Iron overload and tuberculosis: a case for iron chelation therapy.
Cronje L, Bornman L.
Division of Biochemistry, Department of Chemistry and Biochemistry,
University of Johannesburg, Auckland Park, South Africa.
Elevated levels of iron impair immune defence mechanisms, and
specifically the macrophage function of innate immunity. Iron enhances
Mycobacterium tuberculosis infection, M. tuberculosis replication,
progression to clinical disease and death from tuberculosis (TB).
Chelation of iron in individuals with an excessive iron burden may
reduce M. tuberculosis viability and replication, restore host defence
mechanisms and could find application in the prevention and treatment
strategies in settings where both iron overload and TB are prevalent.
The objective of this paper was to summarise recent literature on the
role of iron in TB pathogenesis and to examine the potential of iron
chelation therapy. The literature confirms a key role for iron in
mycobacterial virulence. The ability of chelation to enhance host
effector mechanisms and to inhibit replication of various pathogens
justifies further studies into iron chelation as a potential additive
therapy for TB.
Publication Types:
Review
Review, Tutorial
PMID: 15675543 [PubMed - indexed for MEDLINE]
------------------------------=AD------------------------------=AD---------=
----=AD-------
Who loves ya.
Tom
Jesus Was A Vegetarian! http://jesuswasavegetarian.7h.com
Man Is A Herbivore!
http://pages.ivillage.com/ironjustice/manisaherbivore
DEAD PEOPLE WALKING
http://pages.ivillage.com/ironjustice/deadpeoplewalking
| |
| Arcie Mizelle 2006-03-22, 3:40 pm |
| Excuse me on this one. The cited article does not say that there is an
increased malarial threat from iron supplementation, rather it says there is
an increased malarial threat with iron overload and extremely different
matter. Iron overload is an excess of iron including iron stores. Giving
iron in order to supplement deficiencies is not the same as giving excess
iron to subjects at doses and for a period long enough to create an iron
overload.
You idea may have merit, but you have not chosen a good article to back you
up.
Arcie
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| ironjustice@aol.com 2006-03-22, 3:40 pm |
| >>The cited article does not say that there is an
increased malarial threat from iron supplementation, rather it says
there is
an increased malarial threat with iron overload and extremely different
matter<<
The article states the children are more likely to .. die ..
I said nothing about a .. threat ..
It said .. dying ..
Far past .. threat ..
Who loves ya.
Tom
Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com
Man Is A Herbivore!
http://pages.ivillage.com/ironjustice/manisaherbivore
DEAD PEOPLE WALKING
http://pages.ivillage.com/ironjustice/deadpeoplewalking
Children at risk
of malaria are more likely to die of the disease if they are given
dietary supplements of iron and folic acid,
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