Hepatitis disease - Chronic hepatitis C / iron-restricted diet / liver injury

Author

Chronic hepatitis C / iron-restricted diet / liver injury

ironjustice@aol.com

2006-01-01, 12:56 pm

Hepatogastroenterology. 2002 Mar-Apr;49(44):529-31. Related Articles,
Links

Dietary iron restriction improves aminotransferase levels in chronic
hepatitis C patients.

Iwasa M, Kaito M, Ikoma J, Kobayashi Y, Tanaka Y, Higuchi K, Takeuchi
K, Iwata K, Watanabe S, Adachi Y.

Third Department of Internal Medicine, Mie university School of
Medicine, Edobashi 2-174, Tsu City, Mie 514-8507, Japan.
motoh@clin.medic.mie-u.ac.jp

BACKGROUND/AIMS: It is generally accepted that iron overload plays an
important role in the pathogenesis of liver cell injury in chronic
hepatitis C. The present study was undertaken to evaluate whether
low-iron diet improves liver function tests in patients with chronic
hepatitis C. METHODOLOGY: Seventeen patients with chronic hepatitis C
(13 men and 4 women, 54 +/- 14 years old) that did not respond to, or
were unsuitable for interferon therapy, were enrolled in this study.
All patients had been pretreated with ursodeoxycholic acid for more
than 12 months before the beginning of the study. Dietary iron intake
was restricted to less than 7 mg/day, and the patients were followed up
for 18 months. RESULTS: Mean daily iron intakes, calculated from food
records, were 5.9 and 6.4 mg after 6 and 12 months, respectively. The
mean serum ferritin decreased significantly from 362 ng/mL at entry to
179 ng/mL after 18 months. The serum unsaturated iron binding capacity
level increased significantly from 163 micrograms/dL at entry to 203
micrograms/dL after 18 months. The serum aspartate aminotransferase
decreased significantly from 62 IU/L at entry to 47 IU/L after 18
months, and serum alanine aminotransferase from 68 IU/L at entry to 53
IU/L after 18 months. Serum iron, hepatitis C virus-RNA titer and
platelet count remained unchanged throughout the study. CONCLUSIONS:
These results suggest that iron-restricted diet may be an important
therapeutic modality for improving liver injury in patients with
chronic hepatitis C.

PMID: 11995488 [PubMed - indexed for MEDLINE]

--------------------------------------------------------------------------------

Who loves ya.
Tom

Jesus Was A Vegetarian!
http://jesuswasavegetarian.7h.com

Man Is A Herbivore!
http://pages.ivillage.com/ironjustice/manisaherbivore

DEAD PEOPLE WALKING
http://pages.ivillage.com/ironjustice/deadpeoplewalking

montygram

2006-01-02, 1:08 am

"It is generally accepted..."

I read this all the time. It usually means that original, on point
experiments establishing the claim were never done, or were terribly
flawed, as is the case for "essential fatty acids," for example.
British scientist Harold Hillman wrote an entire book on a few examples
of this. Hep C. is a fabrication. Iron probably plays a major role,
along with lipid peroxidation and exposure to some (if not many)
"pathogenic organisms." The same nonsense is true of "HIV/AIDS," where
the "virus" was never found (even though the technology is available -
has been for a long time - and is not especially expensive), but
instead protein fragments which are just cellular debris have been
declared "markers" of infection. Listen to these people and you are in
deep trouble, as I almost was. I was told to take anti-acid
medication, but determined by my own research that I needed to take
stamach acid and pepsin supplements, which I still do to this day,
several years later, with no signs of the original "disease," no thanks
to "modern medicine." Interestingly, I purchased an old Cycopedia of
Medicine from circa 1920 and that was the only refernce for using
stomach acid supplementation. These "experts" have "gone backwards,"
though the technology has advanced, and creates an image of "great
achievements," depsite the incredible failures of the "war on cancer,"
the "AIDS vaccine" (which is an impossiblity), etc.