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Home > Archive > Nursing > September 2004 > Re: Racketeering (RICO) Charges Filed Against NutraSweet, Dr. Robert Moser of NS, Amer
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Re: Racketeering (RICO) Charges Filed Against NutraSweet, Dr. Robert Moser of NS, Amer
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| Sarah Miller 2004-09-21, 3:25 am |
| kurtullman@yahoo.com (Kurt Ullman) wrote in message news:<Hdp2d.3971$mb6.2793@newsread3.news.atl.earthlink.net>...
[vbcol=seagreen]
CORRECTION THERE ARE NEW RESEARCH FINDINGS!
The articles I listed are very well written articles with scientific
evidence. The suit was filed by the National Justice League not this
whoever you say. You are a rude person, and evidently, very, very
closed minded.
These are the articles again:
Aspartame - Sweetness or Disease?
http://www.dldewey.com/aspar.htm
The $350 Million Lawsuit
http://www.dldewey.com/rico.htm
HERE ARE SOME VERY RECENT STUDIES THAT SHOW ASPARTAME
IS DEADLY TO THE BODY IN MANY WAYS!
THESE ARE ALL ON PUBMED, the URL's are listed as well, they are not
PHONY
as you put it! READ YOU MAY LEARN SOMETHING !!! ! ! ! !
Pharmacol Biochem Behav. 2004 May;78(1):121-7.
Chronic aspartame affects T-maze performance, brain cholinergic
receptors and Na+,K+-ATPase in rats.
http://www.ncbi.nlm.nih.gov/entrez/...t_uids=15159141
Department of Pharmacology, Brody School of Medicine, East Carolina
University, Greenville, NC 27858, USA.
This study demonstrated that chronic aspartame consumption in rats can
lead to altered T-maze performance and increased muscarinic
cholinergic receptor densities in certain brain regions. Control and
treated rats were trained in a T-maze to a particular side and then
periodically tested to see how well they retained the learned
response. Rats that had received aspartame (250 mg/kg/day) in the
drinking water for 3 or 4 months showed a significant increase in time
to reach the reward in the T-maze, suggesting a possible effect on
memory due to the artificial sweetener. Using [(3)H]quinuclidinyl
benzilate (QNB) (1 nM) to label muscarinic cholinergic receptors and
atropine (10(-6) M) to determine nonspecific binding in whole-brain
preparations, aspartame-treated rats showed a 31% increase in receptor
numbers when compared to controls. In aspartame-treated rats, there
was a significant increase in muscarinic receptor densities in the
frontal cortex, midcortex, posterior cortex, hippocampus, hypothalamus
and cerebellum of 80%, 60%, 61%, 65%, 66% and 60%, respectively. The
midbrain was the only area where preparations from aspartame-treated
rats showed a significant increase in Na(+),K(+)-ATPase activity. It
can be concluded from these data that long-term consumption of
aspartame can affect T-maze performance in rats and alter receptor
densities or enzymes in brain.
Magnes Res. 2001 Sep;14(3):189-94. Related Articles, Links
The effect of oral aspartame administration on the balance of
magnesium in the rat.
Kovatsi L, Tsouggas M.
http://www.ncbi.nlm.nih.gov/entrez/...t_uids=11599551
Laboratory of Forensic Medicine & Toxicology, Faculty of Medicine,
Aristotle university of Thessaloniki, Greece. kovatsi@med.auth.gr
The aim of the present work was to determine the effect of aspartame
administration on the excretion of magnesium and its distribution in
the various rat tissues and organs. The present results have shown
that aspartame administration influences the balance of magnesium in
the organism, since in some organs and tissues (heart, lungs, kidneys,
adrenals, jejunum, hair and blood) it is accumulated, while other
organs (liver and testes) are deprived of it. Aspartame administration
also affects the excretion of magnesium from the organism, since it
decreases the concentration of magnesium in both urine and feces.
J Pharmacolofy Science. 2003 Jan;91(1):83-6.
Formaldehyde-induced shrinkage of rat thymocytes.
Nakao H, Umebayashi C, Nakata M, Nishizaki Y, Noda K, Okano Y, Oyama
Y.
Laboratory of Cell Signaling, Faculty of Integrated Arts and Sciences,
The university of Tokushima, Tokushima, Japan.
http://www.ncbi.nlm.nih.gov/entrez/...t_uids=12686735
Laboratory of Cell Signaling, Faculty of Integrated Arts and Sciences,
The university of Tokushima, Tokushima, Japan.
To test the possibility that micromolar formaldehyde, a metabolite of
methanol derived from aspartame, exerts cytotoxicity, its effect on
rat thymocytes was examined under the in vitro condition using a flow
cytometer. Incubation of thymocytes with formaldehyde at 100 micro M
or more for 24 h significantly increased the populations of shrunken
cells and cells with hypodiploid DNA. The peak blood concentration of
methanol in human subjects administered abuse doses of aspartame has
been reported to exceed 2 mg/dL (625 micro M). It would increase the
population of thymocytes undergoing apoptosis if formaldehyde at 100
micro M or more appears in the blood after administration of
aspartame.
Pediatric Neurolgy 2003 Jan;28(1):9-15.
The diet factor in pediatric and adolescent migraine.
Millichap JG, Yee MM.
http://www.ncbi.nlm.nih.gov/entrez/...t_uids=12657413
Division of Neurology, Children's Memorial Hospital, Chicago, Illinois
60614, USA.
Diet can play an important role in the precipitation of headaches in
children and adolescents with migraine. The diet factor in pediatric
migraine is frequently neglected in favor of preventive drug therapy.
The list of foods, beverages, and additives that trigger migraine
includes cheese, chocolate, citrus fruits, hot dogs, monosodium
glutamate, aspartame, fatty foods, ice cream, caffeine withdrawal, and
alcoholic drinks, especially red wine and beer. Underage drinking is a
significant potential cause of recurrent headache in today's
adolescent patients. Tyramine, phenylethylamine, histamine, nitrites,
and sulfites are involved in the mechanism of food intolerance
headache. Immunoglobulin E-mediated food allergy is an infrequent
cause. Dietary triggers affect phases of the migraine process by
influencing release of serotonin and norepinephrine, causing
vasoconstriction or vasodilatation, or by direct stimulation of
trigeminal ganglia, brainstem, and cortical neuronal pathways.
Treatment begins with a headache and diet diary and the selective
avoidance of foods presumed to trigger attacks. A universal migraine
diet with simultaneous elimination of all potential food triggers is
generally not advised in practice. A well-balanced diet is encouraged,
with avoidance of fasting or skipped meals. Long-term prophylactic
drug therapy is appropriate only after exclusion of
headache-precipitating trigger factors, including dietary factors.
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