Psoriasis support - Can this be the case of psoriasis?

Author

Can this be the case of psoriasis?

Cruiser

2005-09-24, 1:41 pm

Crusier: Now this bit is interesting, because B12 and folate are needed for
cell division and DNA synthesys.:

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Thus, in both folate and vitamin B12 deficiency, folate is unavailable to
participate in DNA synthesis. This impairment of DNA synthesis affects the
rapidly dividing cells of the bone marrow earlier than other cells,
resulting in the production of large, immature, hemoglobin-poor red blood
cells.

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Cruiser: I have seen pictures of the defective blood cells compare to
healthy blood cells. They are very reminiscent of the appearance of
psoriatic tissue compared to normal tissue.

Now homocysteine is a growth promotor, so could it be involved in
promoting the growth of psoriatic plaques?

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Second, because homocysteine is a growth promoter, too much of it can
increase the size of smooth muscle cells in artery linings.
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Cruiser: So, you get a combination of accelerated growth of defective cells.

So could that be posriasis?

Cruiser

Cruiser

2005-09-24, 1:41 pm

> Crusier: Now this bit is interesting, because B12 and folate are needed
for
> cell division and DNA synthesys.:
>
> *****************************************
>
> Thus, in both folate and vitamin B12 deficiency, folate is unavailable to
> participate in DNA synthesis. This impairment of DNA synthesis affects the
> rapidly dividing cells of the bone marrow earlier than other cells,
> resulting in the production of large, immature, hemoglobin-poor red blood
> cells.
>
> *****************************************
>
> Cruiser: I have seen pictures of the defective blood cells compare to
> healthy blood cells. They are very reminiscent of the appearance of
> psoriatic tissue compared to normal tissue.
>
> Now homocysteine is a growth promotor, so could it be involved in
> promoting the growth of psoriatic plaques?
>
> *****************************************
> Second, because homocysteine is a growth promoter, too much of it can
> increase the size of smooth muscle cells in artery linings.
> *****************************************
>
> Cruiser: So, you get a combination of accelerated growth of defective
cells.
>
> So could that be posriasis?
>

Now if you add this bit to account for the inflammation?

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http://www.orthomed.com/critical.htm

Free radicals are an almost universal sign of damage to the body. It would
not do for the immune system to be taking off after every foreign
macromolecule that enters into the body. Actually this is what frequently
happens when one is chronically ill for long periods of time. The
immunesystem is activated by the resulting free radicals. All this is a
theory of mine to explain the observed facts but there is experimental
evidence that the affinity of antibodies for their antigen is greater in an
oxidizing redox potential. No one else seems to make anything of that fact.

Fortunately, while ascorbate in massive quantities suppresses this humeral
immunity of antibodies, it augments the cellular immunity of phagocytic
cells. I think it does this both by providing reducing power that initiates
the respiratory burst (by the reduction of molecular oxygen); and by
protecting the white cell from those very radicals the white cell makes in
its vacuoles to kill the pathogens. White cells are unable to continue to
produce these "good" radicals when the radicals leak significantly into the
cytoplasm (then becoming "bad" free radicals) and exceed the ability of
their free radical scavengers to neutralize. All this is useful in the
treatment of autoimmune diseases and allergies butthis is another story.

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Cruiser