| robertke@comcast.net 2004-08-26, 4:07 am |
| Mary:
In Dr.Lerner's research he biopsied heart wall tissue of diagnosed CFS patients and found under the microscope that partially replicated CMV and/or EBV virus particles had disrupted the normal structure of the tissue, i,e., this was the cause of the obse
rved thickening and stiffening of the heart wall and the cause of the heart failure (fatigue). In subsequent research Dr. Lerner found that treatment with the appropriate antiviral drug - Valcyte for CMV and Valtrex for EBV resulted in reversing a signif
icant portion of this damage and a consequent substantial improvement in the patientsexercise performance and endurance.
The infection is active and spreading when the virus is in the bloodstream. The damage is done when the virus gets into body tissues, the heart and elsewhere and tries to replicate.
I would send you a copies of the research to read for yourself, but I've just suffered a hard disk failure and do not know yet whether I still have copies.
Bob Kessler
> In a message dated 8/18/2004 1:41:33 AM Eastern Standard Time,
> robertke@comcast.net writes:
>
>
> Bob,
> I'm confused. If the virus is NOT active and I have antibodies and damage
> from it, what good will taking the antiviral meds do me? Wouldn't the virus
> have to be in an ACTIVE state to get zapped by the meds?
>
> Thanks for your patience.
> Mary
|